Alzheimer disease: Transport protein ABCC1 plays key role in clearing beta-amyloid from brains of mice

Alzheimer disease (AD) is the most common cause of dementia among older people. One of the main features of AD is the presence in the brain of abnormal clumps of the protein fragment beta-amyloid. Researchers have now identified a way to reduce the amount of beta-amyloid in the brains of mice with a disease that models AD, providing hope that a similar approach could benefit patients with this devastating condition.

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