Disruption of cytoskeleton pathways contribute to amyotrophic lateral sclerosis pathogenesis
Sunday, July 15, 2012 - 14:00
in Biology & Nature
A new genetic mutation that causes familial amyotrophic lateral sclerosis (ALS), a fatal neurological disorder also known as Lou Gehrig's Disease, has been identified. Mutations to the profilin gene, which is essential to the growth and development of nerve cell axons, points to defects in a neuron's cytoskeleton structure as a potential common feature among diverse ALS genes.