Finding That Links ALS/Ataxia to Cellular Stress Opens New Approaches for Treatment
Friday, September 7, 2018 - 13:40
in Health & Medicine
S(break)(break)cientists at University of Utah Health report for the first time that a protein, called Staufen1, accumulates in cells of patients suffering from degenerative ataxia or amyotrophic lateral sclerosis (ALS), also known as Lou Gehrig's disease. Depleting the protein from affected mice improved symptoms including motor function. These results suggest that targeting Staufen1 could have therapeutic potential in people. The research are published in Nature Communications